Comment on “Gene Disruption Study Reveals a Nonredundant Role for TRIM21/Ro52 in NF-κB-Dependent Cytokine Expression in Fibroblasts”

Eitemau Tebyg

• Gene disruption study reveals a non-redundant role for TRIM21/Ro52 in NF-κB-dependent cytokine expression in fibroblasts
  gan: Yoshimi, Ryusuke, et al.
  Cyhoeddwyd: (2009)
• Autoantigen TRIM21/Ro52 as a Possible Target for Treatment of Systemic Lupus Erythematosus
  gan: Yoshimi, Ryusuke, et al.
  Cyhoeddwyd: (2012)
• Tripartite Motif (TRIM) 12c, a Mouse Homolog of TRIM5, Is a Ubiquitin Ligase That Stimulates Type I IFN and NF-κB Pathways along with TNFR-Associated Factor 6
  gan: Chang, Tsung-Hsien, et al.
  Cyhoeddwyd: (2015)
• TRIM family proteins and their emerging roles in innate immunity
  gan: Ozato, Keiko, et al.
  Cyhoeddwyd: (2008)
• Clinical and Pathological Roles of Ro/SSA Autoantibody System
  gan: Ryusuke Yoshimi, et al.
  Cyhoeddwyd: (2012-01-01)