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Complement Activation and Toll-Like Receptor-2 Signaling Contribute to Cytokine Production After Renal Ischemia/Reperfusion
The innate immune system causes tissue inflammation and injury after renal ischemia/reperfusion (I/R). The complement system is activated on ischemic tubular epithelial cells (TECs) and induces the cells to produce pro-inflammatory chemokines. TECs also express toll-like receptors (TLRs) -2 and -4....
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| Main Authors: | , , , , , |
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| Format: | Artigo |
| Jezik: | Inglês |
| Izdano: |
2012
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| Teme: | |
| Online dostop: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3405836/ https://ncbi.nlm.nih.gov/pubmed/22750071 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.molimm.2012.05.020 |
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