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Calpastatin overexpression limits calpain-mediated proteolysis and behavioral deficits following traumatic brain injury
Traumatic brain injury (TBI) results in abrupt, initial cell damage leading to delayed neuronal death. The calcium-activated proteases, calpains, are known to contribute to this secondary neurodegenerative cascade. Although the specific inhibitor of calpains, calpastatin, is present within neurons,...
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| Autors principals: | , , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2012
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3392428/ https://ncbi.nlm.nih.gov/pubmed/22572592 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.expneurol.2012.04.022 |
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