Reduced alcohol intake and reward associated with impaired endocannabinoid signaling in mice with a deletion of the glutamate transporter GLAST

A hyperglutamatergic state has been hypothesized to drive escalation of alcohol intake. This hypothesis predicts that an impairment of glutamate clearance through inactivation of the astrocytic glutamate transporter, GLAST (EAAT1), will result in escalation of alcohol consumption. Here, we used mice...

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Detalhes bibliográficos
Main Authors: Karlsson, Rose-Marie, Adermark, Louise, Molander, Anna, Perreau-Lenz, Stephanie, Singley, Erick, Solomon, Matthew, Holmes, Andrew, Tanaka, Kohichi, Lovinger, David M., Spanagel, Rainer, Heilig, Markus
Formato: Artigo
Idioma:Inglês
Publicado em: 2012
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3372600/
https://ncbi.nlm.nih.gov/pubmed/22342743
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neuropharm.2012.01.027
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