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Defective B cell tolerance in adenosine deaminase deficiency is corrected by gene therapy
Adenosine deaminase (ADA) gene defects are among the most common causes of SCID. Restoration of purine metabolism and immune functions can be achieved by enzyme replacement therapy, or more effectively by bone marrow transplant or HSC gene therapy (HSC-GT). However, autoimmune complications and auto...
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| Hoofdauteurs: | , , , , , |
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| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
American Society for Clinical Investigation
2012
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3366410/ https://ncbi.nlm.nih.gov/pubmed/22622038 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI61788 |
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