Myofibroblasts cause heterogeneous Cx43 reduction and are unlikely to be coupled to myocytes in the healing canine infarct

Following myocardial infarction (MI) inflammatory responses transform cardiac fibroblasts to myofibroblasts, which in vitro studies show form heterocellular gap junctions with cardiac myocytes via Connexin43 (Cx43). The ability to form heterocellular junctions in the intact heart and the impact of t...

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Detaylı Bibliyografya
Asıl Yazarlar: Baum, Jennifer R., Long, Biao, Cabo, Candido, Duffy, Heather S.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: American Physiological Society 2012
Konular:
Integrative Cardiovascular Physiology and Pathophysiology
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC3353799/
https://ncbi.nlm.nih.gov/pubmed/22101526
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00498.2011
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