ロード中...
The demise of a TUDOR under stress opens a chromatin link to 53BP1
EMBO J (2012) 31 8, 1865–1878. doi:10.1038/emboj.2012.47 The mechanisms that localise 53BP1 to sites of DNA double-strand breaks (DSBs) have remained elusive, despite this protein's key roles in DNA damage response signalling and repair processes. Recent studies, including the work by Mallette...
保存先:
| 第一著者: | |
|---|---|
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Nature Publishing Group
2012
|
| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3343343/ https://ncbi.nlm.nih.gov/pubmed/22453339 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/emboj.2012.78 |
| タグ: |
タグ追加
タグなし, このレコードへの初めてのタグを付けませんか!
|