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Collecting duct-specific endothelin B receptor knockout increases ENaC activity
Collecting duct (CD)-derived endothelin-1 (ET-1) acting via endothelin B (ETB) receptors promotes Na(+) excretion. Compromise of ET-1 signaling or ETB receptors in the CD cause sodium retention and increase blood pressure. Activity of the epithelial Na(+) channel (ENaC) is limiting for Na(+) reabsor...
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| Huvudupphovsmän: | , , , |
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| Materialtyp: | Artigo |
| Språk: | Inglês |
| Publicerad: |
American Physiological Society
2012
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| Ämnen: | |
| Länkar: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3328904/ https://ncbi.nlm.nih.gov/pubmed/21918182 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpcell.00301.2011 |
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