טוען...
Nucleotides released from Aβ(1–42)-treated microglial cells increase cell migration and Aβ(1–42) uptake through P2Y(2) receptor activation
Amyloid β-protein (Aβ) deposits in brains of Alzheimer’s disease (AD) patients generate proinflammatory cytokines and chemokines that recruit microglial cells to phagocytose Aβ. Nucleotides released from apoptotic cells activate P2Y(2) receptors (P2Y(2)Rs) in macrophages to promote clearance of dead...
שמור ב:
| Main Authors: | , , , , , , , , , |
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| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
2012
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3323761/ https://ncbi.nlm.nih.gov/pubmed/22353164 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1471-4159.2012.07700.x |
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