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Amyloid beta from axons and dendrites reduces local spine number and plasticity
Excessive synaptic loss is thought to be one of the earliest events in Alzheimer’s disease. Amyloid beta (Aβ), a peptide secreted in an activity-modulated manner by neurons, has been implicated in the pathogenesis of Alzheimer’s disease by removing dendritic spines, sites of excitatory synaptic tran...
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| Autors principals: | , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2009
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3310198/ https://ncbi.nlm.nih.gov/pubmed/20037574 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/nn.2476 |
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