Activation of potassium channels contributes to hypoxic injury in proximal tubules.
The mechanisms responsible for the loss of cell potassium during renal ischemia are poorly understood. The present studies examined the hypothesis that potassium channels are activated as an early response to hypoxia and contribute to potassium loss independent from an inhibition of active K+ uptake...
保存先:
| 出版年: | J Clin Invest |
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| 主要な著者: | , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Society for Clinical Investigation
1994
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC330056/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/7989584/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI117592 |
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