Hyperglycemia normalizes insulin-stimulated skeletal muscle glucose oxidation and storage in noninsulin-dependent diabetes mellitus.

The diminished ability of insulin to promote glucose disposal and storage in muscle has been ascribed to impaired activation of glycogen synthase (GS). It is possible that decreased glucose storage could occur as a consequence of decreased glucose uptake, and that GS is impaired secondarily. Muscle...

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Veröffentlicht in:	J Clin Invest
Hauptverfasser:	Kelley, D E, Mandarino, L J
Format:	Artigo
Sprache:	Inglês
Veröffentlicht:	American Society for Clinical Investigation 1990
Schlagworte:	Research Article
Online-Zugang:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC329837/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2123890/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI114935
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