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Silencing of Amyloid Precursor Protein Expression Using a New Engineered Delta Ribozyme
Alzheimer's disease (AD) etiological studies suggest that an elevation in amyloid-β peptides (Aβ) level contributes to aggregations of the peptide and subsequent development of the disease. The major constituent of these amyloid peptides is the 1 to 40–42 residue peptide (Aβ (40−42)) derived fr...
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| 主要な著者: | , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Hindawi Publishing Corporation
2012
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3296272/ https://ncbi.nlm.nih.gov/pubmed/22482079 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1155/2012/947147 |
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