Traumatic brain injury and trichloroethylene exposure interact and produce functional, histological, and mitochondrial deficits

Lignende værker

• Pioglitazone attenuates mitochondrial dysfunction, cognitive impairment, cortical tissue loss, and inflammation following traumatic brain injury
  af: Sauerbeck, Andrew, et al.
  Udgivet: (2010)
• A Mild Traumatic Brain Injury in Mice Produces Lasting Deficits in Brain Metabolism
  af: Lyons, Danielle N., et al.
  Udgivet: (2018)
• Analysis of regional brain mitochondrial bioenergetics and susceptibility to mitochondrial inhibition utilizing a microplate based system
  af: Sauerbeck, Andrew, et al.
  Udgivet: (2011)
• Mitochondrial DNA and traumatic brain injury
  af: Bulstrode, Harry, et al.
  Udgivet: (2014)
• Posttraining Epinephrine Reverses Memory Deficits Produced by Traumatic Brain Injury in Rats
  af: Lorón-Sánchez, Alejandro, et al.
  Udgivet: (2016)