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Cutting Edge: Regulation of TLR4-driven B cell proliferation by RP105 is not B cell-autonomous
Mechanistic understanding of RP105 has been confounded by the fact that this TLR homolog has appeared to have opposing, cell type-specific effects on TLR4 signaling. While RP105 inhibits TLR4-driven signaling in cell lines and myeloid cells, impaired LPS-driven proliferation by B cells from RP105(−/...
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| Autori principali: | , , , , , , , |
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| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
2012
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3288200/ https://ncbi.nlm.nih.gov/pubmed/22291190 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1103282 |
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