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Diminished glucagon suppression after β-cell reduction is due to impaired α-cell function rather than an expansion of α-cell mass
Impaired suppression of glucagon levels after oral glucose or meal ingestion is a hallmark of type 2 diabetes. Whether hyperglucagonemia after a β-cell loss results from a functional upregulation of glucagon secretion or an increase in α-cell mass is yet unclear. CD-1 mice were treated with streptoz...
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| Huvudupphovsmän: | , , , |
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| Materialtyp: | Artigo |
| Språk: | Inglês |
| Publicerad: |
American Physiological Society
2011
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| Ämnen: | |
| Länkar: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3279300/ https://ncbi.nlm.nih.gov/pubmed/21285404 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpendo.00315.2010 |
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