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Excess α-synuclein worsens disease in mice lacking ubiquitin carboxy-terminal hydrolase L1
Mutations in α-synuclein (αSN) and ubiquitin carboxy-terminal hydrolase L1 (UCH-L1) have been linked to familial Parkinson's disease (PD). Physical and functional interactions between these two proteins have been described. Whether they act additively in vivo to influence disease has remained c...
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| Main Authors: | , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Nature Publishing Group
2012
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3278044/ https://ncbi.nlm.nih.gov/pubmed/22355774 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/srep00262 |
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