Increased FcγRII and III expression in synovium and on monocyte derived macrophages of RA-patients results in altered function after immune complex stimulation

Eitemau Tebyg

• Activating FcγRIII determines cartilage destruction during immune complex arthritis but not in the presence of T-cell immunity
  gan: van Lent, PLEM, et al.
  Cyhoeddwyd: (2002)
• The inhibitory receptor FcγRII reduces joint inflammation in immune complex arthritis not only by inhibition of activatory FcγR but also by efficient clearance of immune complexes
  gan: van Lent, PL, et al.
  Cyhoeddwyd: (2003)
• Sustained high expression of Fcγ receptorII (CD32) on dendritic cells (DCs) in patients with rheumatoid arthritis (RA)
  gan: Radstake, TRDJ, et al.
  Cyhoeddwyd: (2002)
• Role of Activatory FcγRI and FcγRIII and Inhibitory FcγRII in Inflammation and Cartilage Destruction during Experimental Antigen-Induced Arthritis
  gan: Van Lent, Peter L. E. M., et al.
  Cyhoeddwyd: (2001)
• Expression of Toll-like receptor (TLR) 2, TLR3, TLR4 and TLR7 is increased in rheumatoid arthritis synovium and regulates cytokine production by dendritic cells upon stimulation of TLR specific pathways
  gan: Roelofs, MF, et al.
  Cyhoeddwyd: (2005)