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Depolarization and decreased surface expression of K(+) channels contribute to NSAID-inhibition of intestinal restitution
Non-steroidal anti-inflammatory drugs (NSAIDs) contribute to gastrointestinal ulcer formation by inhibiting epithelial cell migration and mucosal restitution; however, the drug-affected signaling pathways are poorly defined. We investigated whether NSAID inhibition of intestinal epithelial migration...
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| Main Authors: | , , , , , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado: |
2007
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| Assuntos: | |
| Acceso en liña: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3269908/ https://ncbi.nlm.nih.gov/pubmed/17499219 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bcp.2007.03.030 |
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