Protein kinase CK2, cystic fibrosis transmembrane conductance regulator, and the ΔF508 mutation. F508 deletion disrupts a kinase-binding site. VOLUME 282 (2007) PAGES 10804-10813

Samankaltaisia teoksia

• Inhibition of Protein Kinase CK2 Closes the CFTR Cl(−) Channel, but has no Effect on the Cystic Fibrosis Mutant ΔF508-CFTR
  Tekijä: Treharne, Kate J., et al.
  Julkaistu: (2009)
• Modulation of Protein Kinase CK2 Activity by Fragments of CFTR Encompassing F508 May Reflect Functional Links with Cystic Fibrosis Pathogenesis
  Tekijä: Pagano, Mario A., et al.
  Julkaistu: (2008)
• Understanding protein kinase CK2 mis-regulation upon F508del CFTR expression
  Tekijä: Venerando, Andrea, et al.
  Julkaistu: (2011)
• Answer to the question page 508
  Julkaistu: (2012)
• Cystic fibrosis transmembrane regulator fragments with the Phe(508) deletion exert a dual allosteric control over the master kinase CK2
  Tekijä: Pagano, Mario A., et al.
  Julkaistu: (2010)