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SV40 deletion mutants lacking the 21-bp repeated sequences are viable, but have noncomplementable deficiencies.

We have constructed deletion mutants of simian virus 40 (SV40) lacking the two tandemly repeated copies or all three copies of the 21-bp repeated sequence located in the origin region. The mutants were viable, but had lower infectivities compared to the wild type. The mutant lacking two copies of th...

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Detalles Bibliográficos
Publicado en:Nucleic Acids Res
Main Authors: Hartzell, S W, Yamaguchi, J, Subramanian, K N
Formato: Artigo
Idioma:Inglês
Publicado: Oxford University Press 1983
Acceso en liña:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC325819/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/6298750/
https://ncbi.nlm.nih.govhttps://doi.org/10.1093/nar/11.5.1601
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