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In the clinic: ongoing clinical trials evaluating c-MET-inhibiting drugs
The c-MET (mesenchymal–epithelial transition factor) pathway is dysregulated in many human cancers and promotes tumor growth, invasion and dissemination. The c-MET receptor tyrosine kinase can be activated via gene mutation, gene amplification, protein overexpression and/or a ligand-dependent autocr...
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| Autori principali: | , |
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| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
SAGE Publications
2011
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3225020/ https://ncbi.nlm.nih.gov/pubmed/22128287 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1177/1758834011423403 |
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