Post-traumatic hypoxia exacerbates neurological deficit, neuroinflammation and cerebral metabolism in rats with diffuse traumatic brain injury

समान संसाधन

• Erythropoietin improves motor and cognitive deficit, axonal pathology, and neuroinflammation in a combined model of diffuse traumatic brain injury and hypoxia, in association with upregulation of the erythropoietin receptor
  द्वारा: Hellewell, Sarah C, और अन्य
  प्रकाशित: (2013)
• Post-Traumatic Hypoxia Is Associated with Prolonged Cerebral Cytokine Production, Higher Serum Biomarker Levels, and Poor Outcome in Patients with Severe Traumatic Brain Injury
  द्वारा: Yan, Edwin B., और अन्य
  प्रकाशित: (2014)
• Annexin A2 Deficiency Exacerbates Neuroinflammation and Long-Term Neurological Deficits after Traumatic Brain Injury in Mice
  द्वारा: Liu, Ning, और अन्य
  प्रकाशित: (2019)
• Sensory Cortex Underpinnings of Traumatic Brain Injury Deficits
  द्वारा: Alwis, Dasuni S., और अन्य
  प्रकाशित: (2012)
• Neutrophil extracellular traps exacerbate neurological deficits after traumatic brain injury
  द्वारा: Vaibhav, Kumar, और अन्य
  प्रकाशित: (2020)