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Arrhythmogenic adverse effects of cardiac glycosides are mediated by redox modification of ryanodine receptors
ABSTRACT: The therapeutic use of cardiac glycosides (CGs), agents commonly used in treating heart failure (HF), is limited by arrhythmic toxicity. The adverse effects of CGs have been attributed to excessive accumulation of intracellular Ca(2+) resulting from inhibition of Na(+)/K(+)-ATPase ion tran...
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| Main Authors: | , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Blackwell Science Inc
2011
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3213417/ https://ncbi.nlm.nih.gov/pubmed/21807619 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2011.210005 |
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