Disruption of Nitric Oxide Signaling by Helicobacter pylori Results in Enhanced Inflammation by Inhibition of Heme Oxygenase-1

A strong cellular crosstalk exists between the pathogen Helicobacter pylori and high-output NO production. However, how NO and H. pylori interact to signal in gastric epithelial cells and modulate the innate immune response is unknown. We show that chemical or cellular sources of NO induce the anti-...

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Main Authors: Gobert, Alain P., Asim, Mohammad, Piazuelo, M. Blanca, Verriere, Thomas, Scull, Brooks P., de Sablet, Thibaut, Glumac, Ashley, Lewis, Nuruddeen D., Correa, Pelayo, Peek, Richard M., Chaturvedi, Rupesh, Wilson, Keith T.
פורמט: Artigo
שפה:Inglês
יצא לאור: 2011
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Article
גישה מקוונת:https://ncbi.nlm.nih.gov/pmc/articles/PMC3208050/
https://ncbi.nlm.nih.gov/pubmed/21987660
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1102111
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