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Bypass of glycan-dependent glycoprotein delivery to ERAD by up-regulated EDEM1

Trimming of mannose residues from the N-linked oligosaccharide precursor is a stringent requirement for glycoprotein endoplasmic reticulum (ER)-associated degradation (ERAD). In this paper, we show that, surprisingly, overexpression of ER degradation–enhancing α-mannosidase-like protein 1 (EDEM1) or...

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Detalhes bibliográficos
Main Authors: Ron, Efrat, Shenkman, Marina, Groisman, Bella, Izenshtein, Yana, Leitman, Julia, Lederkremer, Gerardo Z.
Formato: Artigo
Idioma:Inglês
Publicado em: The American Society for Cell Biology 2011
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3204057/
https://ncbi.nlm.nih.gov/pubmed/21917589
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1091/mbc.E10-12-0944
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