Helicobacter pylori Activates Calpain via Toll-Like Receptor 2 To Disrupt Adherens Junctions in Human Gastric Epithelial Cells

Helicobacter pylori is a risk factor for the development of gastritis, gastroduodenal ulcers, and gastric adenocarcinoma. H. pylori-induced disruption of epithelial adherens junctions (AJs) is thought to promote the development of severe disease; however, the mechanisms whereby H. pylori alters AJ s...

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Xehetasun bibliografikoak
Egile Nagusiak: O'Connor, Pamela M., Lapointe, Tamia K., Jackson, Shannon, Beck, Paul L., Jones, Nicola L., Buret, Andre G.
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: American Society for Microbiology 2011
Gaiak:
Cellular Microbiology: Pathogen-Host Cell Molecular Interactions
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC3187258/
https://ncbi.nlm.nih.gov/pubmed/21825064
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/IAI.05109-11
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