Differential requirement for nitric oxide in IGF-1-induced anti-apoptotic, anti-oxidant and anti-atherosclerotic effects

We have shown previously that insulin like-growth factor I (IGF-1) suppressed atherosclerosis in Apoe(−/−) mice and activated endothelial nitric oxide (NO) synthase. To determine whether IGF-1-induced atheroprotection depends on NO, IGF-1- or saline-infused mice were treated with L-NAME, the pan-NO...

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Bibliografske podrobnosti
Main Authors: Sukhanov, Sergiy, Higashi, Yusuke, Shai, Shaw-Yung, Blackstock, Christopher, Galvez, Sarah, Vaughn, Charlotte, Titterington, Jane, Delafontaine, Patrick
Format: Artigo
Jezik:Inglês
Izdano: 2011
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Article
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC3183325/
https://ncbi.nlm.nih.gov/pubmed/21872589
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.febslet.2011.08.029
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