Mitochondrial Ca(2+) Uptake Is Essential for Synaptic Plasticity in Pain

The increase of cytosolic free Ca(2+) ([Ca(2+)](c)) due to NMDA receptor activation is a key step for spinal cord synaptic plasticity by altering cellular signal transduction pathways. We focus on this plasticity as a cause of persistent pain. To provide a mechanism for these classic findings, we re...

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Bibliografiset tiedot
Päätekijät: Kim, Hee Young, Lee, Kwan Yeop, Lu, Ying, Wang, Jigong, Cui, Lian, Kim, Sang Jeong, Chung, Jin Mo, Chung, Kyungsoon
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Society for Neuroscience 2011
Aiheet:
Articles
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC3179262/
https://ncbi.nlm.nih.gov/pubmed/21900577
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.3093-11.2011
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