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Mitochondrial Ca(2+) Uptake Is Essential for Synaptic Plasticity in Pain
The increase of cytosolic free Ca(2+) ([Ca(2+)](c)) due to NMDA receptor activation is a key step for spinal cord synaptic plasticity by altering cellular signal transduction pathways. We focus on this plasticity as a cause of persistent pain. To provide a mechanism for these classic findings, we re...
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| Autori principali: | , , , , , , , |
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| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
Society for Neuroscience
2011
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3179262/ https://ncbi.nlm.nih.gov/pubmed/21900577 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.3093-11.2011 |
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