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DAPK activates MARK1/2 to regulate microtubule assembly, neuronal differentiation, and tau toxicity
Death-associated protein kinase (DAPK) is a key player in several modes of neuronal death/injury and has been implicated in the late-onset Alzheimer's disease (AD). DAPK promotes cell death partly through its effect on regulating actin cytoskeletons. In this study, we report that DAPK inhibits...
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| Main Authors: | , , , , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Nature Publishing Group
2011
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3178434/ https://ncbi.nlm.nih.gov/pubmed/21311567 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/cdd.2011.2 |
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