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Inhibition of the protein kinase MK-2 protects podocytes from nephrotic syndrome-related injury
While mitogen-activated protein kinase (MAPK) activation has been implicated in the pathogenesis of various glomerular diseases, including nephrotic syndrome (NS), its specific role in podocyte injury is not known. We hypothesized that MK-2, a downstream substrate of p38 MAPK, mediates the adverse e...
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| 主要な著者: | , , , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Physiological Society
2011
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3174552/ https://ncbi.nlm.nih.gov/pubmed/21613416 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00661.2010 |
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