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Diverse Roles of TGF-β/Smads in Renal Fibrosis and Inflammation

TGF-β1 has been long considered as a key mediator in renal fibrosis and induces renal scarring largely by activating its downstream Smad signaling pathway. Interestingly, while mice overexpressing active TGF-β1 develop progressive renal injury, latent TGF-β1 plays a protective role in renal fibrosis...

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Detalhes bibliográficos
Autor principal: Lan, Hui Yao
Formato: Artigo
Idioma:Inglês
Publicado em: Ivyspring International Publisher 2011
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3174390/
https://ncbi.nlm.nih.gov/pubmed/21927575
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