Post-Injury Administration of the Mitochondrial Permeability Transition Pore Inhibitor, NIM811, Is Neuroprotective and Improves Cognition after Traumatic Brain Injury in Rats

Mitochondrial dysfunction is known to play a pivotal role in cell death mechanisms following traumatic brain injury (TBI). N-methyl-4-isoleucine-cyclosporin (NIM811), a non-immunosuppressive cyclosporin A (CsA) analog, inhibits the mitochondrial permeability transition pore (mPTP) and has been shown...

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Detaylı Bibliyografya
Asıl Yazarlar: Readnower, Ryan D., Pandya, Jignesh D., McEwen, Melanie L., Pauly, James R., Springer, Joseph E., Sullivan, Patrick G.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Mary Ann Liebert, Inc. 2011
Konular:
Original Articles
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC3172877/
https://ncbi.nlm.nih.gov/pubmed/21875332
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1089/neu.2011.1755
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