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Endogenous Prolactin Generated During Peripheral Inflammation Contributes to Thermal Hyperalgesia
Prolactin (PRL) is a hormone and a neuromodulator. PRL sensitizes TRPV1 responses in sensory neurons, but it is not clear whether peripheral inflammation results in the release of endogenous PRL, or whether endogenous PRL is capable of acting as an inflammatory mediator in a sex-dependent manner. To...
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| 主要な著者: | , , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2011
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| 主題: | |
| オンライン・アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3168702/ https://ncbi.nlm.nih.gov/pubmed/21777304 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1460-9568.2011.07788.x |
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