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Endogenous Prolactin Generated During Peripheral Inflammation Contributes to Thermal Hyperalgesia

Prolactin (PRL) is a hormone and a neuromodulator. PRL sensitizes TRPV1 responses in sensory neurons, but it is not clear whether peripheral inflammation results in the release of endogenous PRL, or whether endogenous PRL is capable of acting as an inflammatory mediator in a sex-dependent manner. To...

Täydet tiedot

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Bibliografiset tiedot
Päätekijät: Scotland, Phoebe E., Patil, Mayur, Belugin, Sergei, Henry, Michael A., Goffin, Vincent, Hargreaves, Kenneth M., Akopian, Armen N.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: 2011
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC3168702/
https://ncbi.nlm.nih.gov/pubmed/21777304
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1460-9568.2011.07788.x
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