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A ribosomal function is necessary for efficient splicing of the T4 phage thymidylate synthase intron in vivo

Splicing of the group I intron of the T4 thymidylate synthase (td) gene was uncoupled from translation by introducing stop codons in the upstream exon. This resulted in severe splicing deficiency in vivo. Overexpression of a UGA suppressor tRNA partially rescued splicing, suggesting that this in vit...

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Detaylı Bibliyografya
Asıl Yazarlar: Semrad, Katharina, Schroeder, Renée
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Cold Spring Harbor Laboratory Press 1998
Konular:
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC316773/
https://ncbi.nlm.nih.gov/pubmed/9573049
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