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Endothelin-1 contributes to increased NFATc3 activation by chronic hypoxia in pulmonary arteries
Chronic hypoxia (CH) activates the Ca(2+)-dependent transcription factor nuclear factor of activated T cells isoform c3 (NFATc3) in mouse pulmonary arteries. However, the mechanism of this response has not been explored. Since we have demonstrated that NFATc3 is required for CH-induced pulmonary art...
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主要な著者: | , , , , |
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フォーマット: | Artigo |
言語: | Inglês |
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American Physiological Society
2011
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オンライン・アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3154561/ https://ncbi.nlm.nih.gov/pubmed/21525433 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpcell.00029.2011 |
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