Parathyroid Hormone (PTH)–Induced Bone Gain Is Blunted in SOST Overexpressing and Deficient Mice

Intermittent parathyroid hormone (PTH) treatment is a potent bone anabolic principle that suppresses expression of the bone formation inhibitor Sost. We addressed the relevance of Sost suppression for PTH-induced bone anabolism in vivo using mice with altered Sost gene dosage. Six-month-old Sost ove...

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Main Authors: Kramer, Ina, Loots, Gabriela G, Studer, Anne, Keller, Hansjoerg, Kneissel, Michaela
Formáid: Artigo
Teanga:Inglês
Foilsithe: Wiley Subscription Services, Inc., A Wiley Company 2010
Ábhair:
Original Article
Rochtain Ar Líne:https://ncbi.nlm.nih.gov/pmc/articles/PMC3153379/
https://ncbi.nlm.nih.gov/pubmed/19594304
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1359/jbmr.090730
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