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Mammalian achaete–scute and atonal homologs regulate neuronal versus glial fate determination in the central nervous system
Whereas vertebrate achaete–scute complex (as-c) and atonal (ato) homologs are required for neurogenesis, their neuronal determination activities in the central nervous system (CNS) are not yet supported by loss-of-function studies, probably because of genetic redundancy. Here, to address this proble...
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| 主要な著者: | , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Oxford University Press
2000
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC314003/ https://ncbi.nlm.nih.gov/pubmed/11032813 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/emboj/19.20.5460 |
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