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Mammalian achaete–scute and atonal homologs regulate neuronal versus glial fate determination in the central nervous system
Whereas vertebrate achaete–scute complex (as-c) and atonal (ato) homologs are required for neurogenesis, their neuronal determination activities in the central nervous system (CNS) are not yet supported by loss-of-function studies, probably because of genetic redundancy. Here, to address this proble...
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| Yayımlandı: | EMBO J |
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| Asıl Yazarlar: | , , , , |
| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
Nature Publishing Group
2000
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC314003/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/11032813/ https://ncbi.nlm.nih.govhttps://doi.org/10.1093/emboj/19.20.5460 |
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