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Prostaglandins do not contribute to the nitric oxide-mediated compensatory vasodilation in hypoperfused exercising muscle

We tested the hypothesis that 1) prostaglandins (PGs) contribute to compensatory vasodilation in contracting human forearm subjected to acute hypoperfusion, and 2) the combined inhibition of PGs and nitric oxide would attenuate the compensatory vasodilation more than PG inhibition alone. In separate...

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Detaylı Bibliyografya
Asıl Yazarlar: Casey, Darren P., Joyner, Michael J.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: American Physiological Society 2011
Konular:
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC3129921/
https://ncbi.nlm.nih.gov/pubmed/21536852
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00222.2011
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