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Prostaglandins do not contribute to the nitric oxide-mediated compensatory vasodilation in hypoperfused exercising muscle

We tested the hypothesis that 1) prostaglandins (PGs) contribute to compensatory vasodilation in contracting human forearm subjected to acute hypoperfusion, and 2) the combined inhibition of PGs and nitric oxide would attenuate the compensatory vasodilation more than PG inhibition alone. In separate...

Täydet tiedot

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Bibliografiset tiedot
Päätekijät: Casey, Darren P., Joyner, Michael J.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: American Physiological Society 2011
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC3129921/
https://ncbi.nlm.nih.gov/pubmed/21536852
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00222.2011
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