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Prostaglandins do not contribute to the nitric oxide-mediated compensatory vasodilation in hypoperfused exercising muscle

We tested the hypothesis that 1) prostaglandins (PGs) contribute to compensatory vasodilation in contracting human forearm subjected to acute hypoperfusion, and 2) the combined inhibition of PGs and nitric oxide would attenuate the compensatory vasodilation more than PG inhibition alone. In separate...

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Autores principales: Casey, Darren P., Joyner, Michael J.
Formato: Artigo
Lenguaje:Inglês
Publicado: American Physiological Society 2011
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Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC3129921/
https://ncbi.nlm.nih.gov/pubmed/21536852
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00222.2011
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