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Prostaglandins do not contribute to the nitric oxide-mediated compensatory vasodilation in hypoperfused exercising muscle

We tested the hypothesis that 1) prostaglandins (PGs) contribute to compensatory vasodilation in contracting human forearm subjected to acute hypoperfusion, and 2) the combined inhibition of PGs and nitric oxide would attenuate the compensatory vasodilation more than PG inhibition alone. In separate...

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Bibliografiska uppgifter
Huvudupphovsmän: Casey, Darren P., Joyner, Michael J.
Materialtyp: Artigo
Språk:Inglês
Publicerad: American Physiological Society 2011
Ämnen:
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC3129921/
https://ncbi.nlm.nih.gov/pubmed/21536852
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00222.2011
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