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Contribution of adenosine to compensatory dilation in hypoperfused contracting human muscles is independent of nitric oxide
We previously demonstrated that nitric oxide (NO) contributes to compensatory vasodilation in the contracting human forearm subjected to acute hypoperfusion. We examined the potential role of an adenosine-NO interaction to this response in 17 male subjects (25 ± 2 yr). In separate protocols subjects...
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| Autors principals: | , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
American Physiological Society
2011
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3098657/ https://ncbi.nlm.nih.gov/pubmed/21292838 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/japplphysiol.00836.2010 |
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