טוען...
Loss of JNK2 increases intestinal tumor susceptibility in Apc1638+/− mice with dietary modulation
A recent study has shown that c-Jun NH2-terminal kinases (JNKs) 2 interacts with and inhibits β-catenin signaling in vitro. To determine the role of genetic interaction between JNK2 and β-catenin in vivo and to elucidate JNK2-mediated intestinal carcinogenesis, we crossed the JNK2(−/−) mice with Apc...
שמור ב:
| Main Authors: | , , , |
|---|---|
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
Oxford University Press
2011
|
| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3105581/ https://ncbi.nlm.nih.gov/pubmed/21183606 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/carcin/bgq275 |
| תגים: |
הוספת תג
אין תגיות, היה/י הראשונ/ה לתייג את הרשומה!
|