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Loss of JNK2 increases intestinal tumor susceptibility in Apc1638+/− mice with dietary modulation
A recent study has shown that c-Jun NH2-terminal kinases (JNKs) 2 interacts with and inhibits β-catenin signaling in vitro. To determine the role of genetic interaction between JNK2 and β-catenin in vivo and to elucidate JNK2-mediated intestinal carcinogenesis, we crossed the JNK2(−/−) mice with Apc...
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| 主要な著者: | , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Oxford University Press
2011
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3105581/ https://ncbi.nlm.nih.gov/pubmed/21183606 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/carcin/bgq275 |
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