Deficient repair of the transcribed strand of active genes in Cockayne's syndrome cells.

Ítems similars

• The genetic defect in Cockayne syndrome is associated with a defect in repair of UV-induced DNA damage in transcriptionally active DNA.
  per: Venema, J, et al.
  Publicat: (1990)
• Xeroderma pigmentosum complementation group C cells remove pyrimidine dimers selectively from the transcribed strand of active genes.
  per: Venema, J, et al.
  Publicat: (1991)
• Preferential repair of ionizing radiation-induced damage in the transcribed strand of an active human gene is defective in Cockayne syndrome.
  per: Leadon, S. A., et al.
  Publicat: (1993)
• The sensitivity of Cockayne's syndrome cells to DNA-damaging agents is not due to defective transcription-coupled repair of active genes.
  per: van Oosterwijk, M F, et al.
  Publicat: (1996)
• The residual repair capacity of xeroderma pigmentosum complementation group C fibroblasts is highly specific for transcriptionally active DNA.
  per: Venema, J, et al.
  Publicat: (1990)