Early detachment of neuromuscular junction proteins in ALS mice with SODG93A mutation

The transgenic animals with mutant copper/zinc superoxide dismutase (SOD1) DNA develop paralytic motor neuron disease resembling human amyotrophic lateral sclerosis (ALS) patients and are commonly used as models for ALS. In the transgenic (Tg) mice with the G93A mutation of the human SOD1 gene SOD1(...

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Autores principales: Narai, Hisashi, Manabe, Yasuhiro, Nagai, Makiko, Nagano, Isao, Ohta, Yasuyuki, Murakami, Tetsuro, Takehisa, Yasushi, Kamiya, Tatsushi, Abe, Koji
Formato: Artigo
Lenguaje:Inglês
Publicado: PAGEPress Publications 2009
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Article
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC3093225/
https://ncbi.nlm.nih.gov/pubmed/21577353
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4081/ni.2009.e16
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