Early detachment of neuromuscular junction proteins in ALS mice with SODG93A mutation

The transgenic animals with mutant copper/zinc superoxide dismutase (SOD1) DNA develop paralytic motor neuron disease resembling human amyotrophic lateral sclerosis (ALS) patients and are commonly used as models for ALS. In the transgenic (Tg) mice with the G93A mutation of the human SOD1 gene SOD1(...

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Main Authors: Narai, Hisashi, Manabe, Yasuhiro, Nagai, Makiko, Nagano, Isao, Ohta, Yasuyuki, Murakami, Tetsuro, Takehisa, Yasushi, Kamiya, Tatsushi, Abe, Koji
Formato: Artigo
Idioma:Inglês
Publicado em: PAGEPress Publications 2009
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3093225/
https://ncbi.nlm.nih.gov/pubmed/21577353
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4081/ni.2009.e16
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