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Neural Mechanisms of Angiotensin II–Salt Hypertension: Implications for Therapies Targeting Neural Control of the Splanchnic Circulation

Chronically elevated plasma angiotensin II (AngII) causes a salt-sensitive form of hypertension that is associated with a differential pattern of peripheral sympathetic outflow. This “AngII-salt sympathetic signature” is characterized by a transient reduction in sympathetic nervous system activity (...

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Autors principals: Osborn, John W., Fink, Gregory D., Kuroki, Marcos T.
Format: Artigo
Idioma:Inglês
Publicat: 2011
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Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC3076522/
https://ncbi.nlm.nih.gov/pubmed/21298369
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s11906-011-0188-9
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