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Neural Mechanisms of Angiotensin II–Salt Hypertension: Implications for Therapies Targeting Neural Control of the Splanchnic Circulation
Chronically elevated plasma angiotensin II (AngII) causes a salt-sensitive form of hypertension that is associated with a differential pattern of peripheral sympathetic outflow. This “AngII-salt sympathetic signature” is characterized by a transient reduction in sympathetic nervous system activity (...
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| Main Authors: | , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2011
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3076522/ https://ncbi.nlm.nih.gov/pubmed/21298369 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s11906-011-0188-9 |
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