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Intestinal epithelial cells as producers but not targets of chronic TNF suffice to cause murine Crohn-like pathology
TNF plays a crucial role in the pathogenesis of Crohn disease. Dysregulated TNF production in mice that bear the genetic deletion of the TNF AU-rich regulatory elements (ARE) (Tnf(ΔARE/+) mice) results in TNF receptor I (TNFRI)-dependent spontaneous Crohn-like pathology. Current concepts consider in...
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| Main Authors: | , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
National Academy of Sciences
2011
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3069201/ https://ncbi.nlm.nih.gov/pubmed/21402942 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1007811108 |
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